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Download PDF by Professor John Bond, Dr Sheila Peace, Freya Dittmann-Kohli,: Ageing in Society, 3rd Edition

By Professor John Bond, Dr Sheila Peace, Freya Dittmann-Kohli, Gerben Westerhof

ISBN-10: 1412900190

ISBN-13: 9781412900195

ISBN-10: 1412900204

ISBN-13: 9781412900201

ISBN-10: 1848607245

ISBN-13: 9781848607248

The 3rd variation of this well known and widely-used textual content presents a complete advent to the examine of getting older, exploring the main behavioral and social technological know-how theories, thoughts, and strategies. This re-creation of getting old in Society has been broadly rewritten and displays new tendencies in eu gerontology, incorporating contemporary advancements in thought and study from foreign and interdisciplinary views.

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Additional info for Ageing in Society, 3rd Edition

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Since their pathophysiology is complex, each of the sufficient causes of death consists of a large number of component causes. At puberty, when mortality risk is lowest, only a few component causes are present. With increasing age, more component causes will have accumulated. The sufficient causes that together constitute the different potential causes of death are built up step by step throughout life. Every new component cause increases the chance that one of the sufficient causes is completed.

Qxd 1/9/2007 11:39 AM 24 Page 24 AGEING IN SOCIETY DNA through (i) damage to the chromosomal DNA of the cell nucleus resulting in impaired gene function, (ii) damage to telomeres – the protective DNA structures that appear to ‘cap’ the ends of chromosomes (analogous to the plastic tips of shoelaces), and (iii) damage to the DNA that exists within the cell’s energygenerating organelles, the mitochondria, resulting in impaired energy production. Damage to DNA is particularly likely to play a role in the lifelong accumulation of molecular damage within cells, since damage to DNA can readily result in permanent alteration of the cell’s DNA sequence.

Grube and Bürkle (1992) discovered a strong, positive correlation of PARP activity with the species lifespan, cells from long-lived species having higher levels of PARP activity than cells from short-lived species. , 1998). , 2002). This is due to the absence of the enzyme telomerase, which is normally expressed only in germ cells (in testis and ovary) and in certain adult stem cells. Some have suggested that in dividing somatic cells telomeres act as an intrinsic ‘division counter’, perhaps to protect us against runaway cell division as happens in cancer.

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Ageing in Society, 3rd Edition by Professor John Bond, Dr Sheila Peace, Freya Dittmann-Kohli, Gerben Westerhof

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